Author

M. M. Meyer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:bacteria (other)· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Organism:chicken / avian· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

I. Warrier· 2 shared T. van Opijnen· 2 shared A. Hazery· 1 shared A. Michalik· 1 shared A. Perry· 1 shared C. Simon· 1 shared C. Veloso· 1 shared H. Echlin· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersMicrobiology

I. Warrier

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Evaluation of the fitness benefit conferred by RNA cis-regulators to Streptococcus pneumoniae during infection

I. Warrier, A. Perry, S. M. Hubbell, M. Eichelman, T. van Opijnen, M. M. Meyer

This study evaluates the fitness benefit of RNA cis-regulators in Streptococcus pneumoniae during infection, finding that mis-regulation significantly reduces fitness in vivo.

bioRxiv · Dec 8, 2023Microbiology

Comparative metatranscriptomics of periodontitis supports a common polymicrobial shift in metabolic function and identifies novel putative disease-associated ncRNAs

N. Ram Mohan, M. M. Meyer

This study identifies common metabolic shifts and novel non-coding RNAs in periodontitis through comparative metatranscriptomics of three datasets.

Frontiers in Microbiology · Apr 9, 2020Peer-reviewedMicrobiology

The Transcriptional landscape of Streptococcus pneumoniae TIGR4 reveals a complex operon architecture and abundant riboregulation critical for growth and virulence

I. Warrier, N. Ram-Mohan, Z. Zhu, A. Hazery, H. Echlin, J. Rosch, M. M. Meyer, T. van Opijnen

bioRxiv · Nov 8, 2018Microbiology

Changes in endosymbiont complexity drive host-level compensatory adaptations in cicadas

M. A. Campbell, P. Łukasik, M. M. Meyer, M. Buckner, C. Simon, C. Veloso, A. Michalik, J. P. McCutcheon

The paper shows that cicadas with more complex Hodgkinia endosymbiont lineages increase the number of Hodgkinia cells transmitted to eggs to ensure full complement transmission.

bioRxiv · Jun 22, 2018Evolutionary Biology