Author

MA Cousin

Institute for Stem Cell Biology and Regenerative Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:rat· 2 Research Focus:cell type annotation· 1 Assay:electronic health records· 1

Frequent Co-Authors

Peter C. KindiD· 2 shared Alfredo Gonzalez‐SulseriD· 1 shared Daisy Arkell· 1 shared Danai KatsanevakiiD· 1 shared David J. A. WyllieiD· 1 shared E Perkins· 1 shared Elizabeth C. DavenportiD· 1 shared Emily K. OsterweiliD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Peter C. KindORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Enhanced hippocampal LTP but typical NMDA receptor and AMPA receptor function in a novel rat model of CDKL5 deficiency disorder

Laura Simões de Oliveira, Heather O’Leary, MS Nawaz, Rita M. Loureiro, Elizabeth C. Davenport, Paul S. Baxter, SR Ribeiro dos Louros, OR Dando, E Perkins, Emily K. Osterweil, GE Hardingham, MA Cousin, S Chattarji, SA Booker, TA Benke, , Peter C. Kind

This study investigates synaptic function in a Cdkl5 KO rat model of CDKL5 deficiency disorder, finding normal NMDA and AMPA receptor function but altered synaptic density and plasticity.

bioRxiv · Jun 29, 2022 · PreprintNeuroscience

Heterozygous deletion of SYNGAP enzymatic domains in rats causes selective learning, social and seizure phenotypes

Danai Katsanevaki, SM Till, Ingrid Buller‐Peralta, TC. Watson, MS. Nawaz, Daisy Arkell, S. Tiwari, Vijayakumar Kapgal, Shyam S. Biswal, JAB. Smith, NJ. Anstey, Lindsay A. M. Mizen, Nicholas Perentos, MW. Jones, MA Cousin, , Peter C. Kind

This study creates a rat model with SYNGAP enzymatic domain deletion, showing learning, social, and seizure deficits in heterozygous animals.

bioRxiv · Oct 14, 2020 · PreprintNeuroscience