Author

Madhurima Das

Boston University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:simulation· 1

Frequent Co-Authors

Afra Panahi· 1 shared Ali ShilatifardiD· 1 shared Andrew G. Burwash· 1 shared Antoine DürrbachiD· 1 shared Antoine Pierre BréziniD· 1 shared Benjamin Charles Howard· 1 shared Bin Zheng· 1 shared Caroline Beugnet· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMolecular Biology

Afra Panahi

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2 papers

Recent·Most cited

BRD2 bridges TFIID and histone acetylation to promote transcriptional initiation

Bin Zheng, Ruxuan Qiu, Sarah R. Gold, Marta Iwanaszko, Yuki Aoi, Benjamin Charles Howard, Madhurima Das, Ali Shilatifard

This study shows BRD2 promotes transcriptional initiation via bromodomain-dependent binding to acetylated histones and recruits TFIID, while BRD4 mediates elongation through CDK9 recruitment.

bioRxiv · Sep 20, 2025 · PreprintBiochemistry

Molecular basis for a novel systemic form of human hereditary apoA-I amyloidosis with vision loss

Isabel M Morgado, Pierre-Raphaël Rothschild, Afra Panahi, Jean‐Claude Aldigier, Andrew G. Burwash, Madhurima Das, Magali Colombat, Thierry Frouget, Jean Philippe Rerolle, François Paraf, Nathalie C. Rioux-Leclercq, Jean‐Michel Goujon, Caroline Beugnet, Antoine Dürrbach, Didier Samuel, , Sophie Valleix

The paper identifies a novel form of hereditary apoA-I amyloidosis caused by Glu34Lys mutation, revealing structural changes that lead to amyloid formation and vision loss.

bioRxiv · Jun 22, 2018 · PreprintMolecular Biology