Author

Magali Colombat

Institut universitaire du cancer de Toulouse OncopoleORCID 0000-0001-8317-2868Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:simulation· 1 Approach:statistical model· 1

Frequent Co-Authors

Afra Panahi· 1 shared Alessio LampisiD· 1 shared Andrew G. Burwash· 1 shared Antoine DürrbachiD· 1 shared Antoine Pierre BréziniD· 1 shared Camille CoheniD· 1 shared Caroline Beugnet· 1 shared Christophe SiraciD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMolecular Biology

Afra Panahi

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2 papers

Recent·Most cited

PathologybioRxivPreprintDec 23, 2025

Accelerated Apolipoprotein A-II senile amyloidosis in a PAI-1 (plasminogen activator inhibitor 1) knock-out model

Gilles Roussine Codo, Pauline Duchatelet, Gemma Martinez-Rivas…·Christophe Sirac

This study shows PAI-1 knockout mice develop systemic AApoA-II amyloidosis at intermediate age, with amyloid deposits in multiple organs and urinary albumin as a biomarker, highlighting potential risks of PAI-1 inhibition in amyloidosis.

DOI 10.64898/2025.12.18.695159

mouseexperimental assay

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Molecular BiologybioRxivPreprintJun 22, 2018

Molecular basis for a novel systemic form of human hereditary apoA-I amyloidosis with vision loss

Isabel M Morgado, Pierre-Raphaël Rothschild, Afra Panahi…·Sophie Valleix

The paper identifies a novel form of hereditary apoA-I amyloidosis caused by Glu34Lys mutation, revealing structural changes that lead to amyloid formation and vision loss.

DOI 10.1101/354001

humanproteomics

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