Author

Martin Lee

Edinburgh Cancer Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:mouse· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Alexander von Kriegsheim· 1 shared Andreea Gradinaru· 1 shared Ann P. WheeleriD· 1 shared Anthony TumberiD· 1 shared Chartinun Chutoe· 1 shared Chinmayi PednekariD· 1 shared Chris BakaliD· 1 shared Christopher J. SchofieldiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

Alexander von Kriegsheim

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Multi-Site Reproducibility Study of 3D High-Content Analysis with Dual-View Oblique Plane Microscopy

Yuriy Alexandrov, Mar Arias-García, Chris Bakal, Eduard Batlle, Vicky Bousgouni, Neil O. Carragher, Julien Colombelli, Jayne Culley, Nathan Curry, Lucas Dent, Christopher W. Dunsby, Liuba Dvinskikh, Edwin Garcia, Nikolaos-Nikiforos Giakoumakis, Nils Gustafsson, , Theresa Suckert

This study assesses the reproducibility of a 3D spheroid-based assay across four institutes using dual-view oblique plane microscopy and finds high consistency in treatment effects with low technical noise.

bioRxiv · Jul 8, 2026 · PreprintBioengineering

Kindlin-1 loss disrupts vascular and extracellular matrix organisation to sustain hypoxia in cutaneous squamous cell carcinoma

David Hardman, Giovana Carrasco, Martin Lee, Muhammad Furqan, Romain Enjalbert, Valerie G. Brunton, Miguel O. Bernabéu

This study shows that Kindlin-1 loss in cutaneous squamous cell carcinoma disrupts ECM-vascular organization, leading to hypoxia and tumor progression.

bioRxiv · Jun 3, 2026 · PreprintCancer Biology

JMJD5 regulates metabolism by hydroxylating ISY1, and regulating the Arginine Methyltransferase PRMT6

Zaid Khan, Chartinun Chutoe, Jair Gonzalez Marques Júnior, Edward J. Jarman, Andreea Gradinaru, Anthony Tumber, Eidarus Salah, Martin Lee, Haya Al Siyabi, Philippe Emmanuel Gautier, Chinmayi Pednekar, Ann P. Wheeler, Christopher J. Schofield, Luke Boulter, Alexander von Kriegsheim

This paper identifies JMJD5-ISY1-PRMT6 as a pathway regulating metabolism by hydroxylating ISY1 to inhibit PRMT6, showing its role in liver cancer and potential as a therapeutic target.

bioRxiv · Sep 27, 2025 · PreprintCell Biology