Author

N. Scholz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

J. D. F. Licchesi· 2 shared C. Lindon· 1 shared F. A. Siebzehnrubl· 1 shared J.-P. Warnke· 1 shared K. Eschrich· 1 shared M. Bigl· 1 shared N. Vaughan· 1 shared R. Kessler· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 14 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

J. D. F. Licchesi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

HECTD1 is both a positive regulator and substrate of caspase-3 activity during apoptotic cell death

N. Scholz, F. A. Siebzehnrubl, J. D. F. Licchesi

This study identifies HECTD1 as a caspase-3 substrate during apoptosis, showing cleavage does not affect its HECT domain function and suggests a feedback loop between caspase-3 and HECTD1.

bioRxiv · Feb 17, 2023 · PreprintCell Biology

The E3 ubiquitin ligase HECTD1 contributes to cell cycle progression through an effect on mitosis

N. Vaughan, N. Scholz, C. Lindon, J. D. F. Licchesi

This study shows that HECTD1 ubiquitin ligase activity promotes cell cycle progression through its role in mitosis by affecting chromosome alignment and spindle assembly checkpoint activity.

bioRxiv · Dec 17, 2021 · PreprintCell Biology

Functional diversity of PFKFB3 splice variants in glioblastomas

U. Heydasch, R. Kessler, J.-P. Warnke, K. Eschrich, N. Scholz, M. Bigl

This study investigates the functional roles of PFKFB3 splice variants in glioblastomas, finding variant-specific effects on cell viability and proliferation, with PFKFB3-4 promoting and PFKFB3-5 inhibiting tumor growth.

bioRxiv · Oct 9, 2020 · PreprintCancer Biology