Author

Nadia Illma

Howard Hughes Medical Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Aliyu Jabir Muhammad· 1 shared Berkan KanmaziD· 1 shared Chun‐Xia YiiD· 1 shared Daniel Hüebschmann· 1 shared Daniel N. KazdaliD· 1 shared Derya S. Arslan· 1 shared Dick Frans SwaabiD· 1 shared Elena G. BochukovaiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Aliyu Jabir Muhammad

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1 paper

Recent·Most cited

Magel2 deficiency drives microglial synaptic engulfment and alters neuron-microglia interactions in Prader-Willi syndrome

Jemima Helen, Moritz Claudius Wimmer, Berkan Kanmaz, Tim Schubert, Ingrid Camila Possa‐Paranhos, Felipe Corrêa‐da‐Silva, Yuval Podpecan, Aliyu Jabir Muhammad, Eva M. Eisemann, Nadia Illma, Derya S. Arslan, Megan Iminitoff, Franziska Schommer, Henning Fröhlich, Felix Franke, , Ferdinand Althammer

This study shows that Magel2 deficiency in a mouse model of Prader-Willi syndrome causes microglial structural changes and increased synaptic engulfment, altering neuron-microglia interactions and hypothalamic circuit function.

bioRxiv · Sep 30, 2026 · PreprintNeuroscience