Author

Eva M. Eisemann

University Hospital Heidelberg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Aliyu Jabir Muhammad· 1 shared Berkan KanmaziD· 1 shared Chun‐Xia YiiD· 1 shared Daniel Hüebschmann· 1 shared Daniel N. KazdaliD· 1 shared Derya S. Arslan· 1 shared Dick Frans SwaabiD· 1 shared Elena G. BochukovaiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Aliyu Jabir Muhammad

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1 paper

Recent·Most cited

NeurosciencebioRxivPreprintSep 30, 2026

Magel2 deficiency drives microglial synaptic engulfment and alters neuron-microglia interactions in Prader-Willi syndrome

Jemima Helen, Moritz Claudius Wimmer, Berkan Kanmaz…·Ferdinand Althammer

This study shows that Magel2 deficiency in a mouse model of Prader-Willi syndrome causes microglial structural changes and increased synaptic engulfment, altering neuron-microglia interactions and hypothalamic circuit function.

DOI 10.64898/2026.09.30.755583

mousebulk RNA-seq

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