Author

Nicholas Bodenstein

TUM Klinikum

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1

Frequent Co-Authors

Julius Clemens FischeriD· 2 shared Markus TschurtschenthaleriD· 2 shared Nicole A SchmidiD· 2 shared Anantharamanan RajamaniiD· 1 shared Antonio Enrico ZauritoiD· 1 shared Aritra MahapatraiD· 1 shared Ashley D. SandersiD· 1 shared Björn KonukiewitziD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

Julius Clemens FischerORCID

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2 papers

Recent·Most cited

Distinct colorectal cancer genotypes shape microbial ecosystems and reveal stage-specific microbiota dependencies

Valentina Brunner, Nicholas Bodenstein, Antonio Enrico Zaurito, M Silva, Frederic Saab, Fabian Springer, Fabio Boniolo, Moritz Jesinghaus, Anantharamanan Rajamani, Aritra Mahapatra, Tanja Groll, Nicole A Schmid, Rupert Oellinger, Chen Meng, Sabine Schwamberger, , Markus Tschurtschenthaler

This study shows that different colorectal cancer genotypes create distinct microbial ecosystems and that microbiota dependence decreases as tumors progress.

bioRxiv · Jul 7, 2026 · PreprintCancer Biology

Interferon-independent STING enforces epithelial genome-integrity checkpoint to restrain tumor evolution

Xiang Hang, Martina Macino, Clemens Woitaske-Proske, Nicholas Bodenstein, J P Bernardes, Florian Tran, Nicole A Schmid, Gabriela Rios Martini, Felix Wottawa, J Bornhaeuser, Nassim Kakavand, Oliver Dyck Dionisi, Silke van den Bossche, Guang Yang, Qicong Wu, , Konrad Aden

This study shows that STING acts as a genome-integrity checkpoint in epithelial cells, independent of interferon, and its loss leads to chromosomal instability and tumor development.

bioRxiv · Mar 25, 2026 · PreprintCancer Biology