Author

O. Babur

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Korkut· 2 shared B. Bozorgui· 2 shared X. Li· 2 shared Z. Dereli· 2 shared A. Luna· 1 shared C. Sander· 1 shared E. K. Kong· 1 shared G. Mills· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCancer Biology

A. Korkut

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2 papers

Recent·Most cited

A prognostic matrix code defines functional glioblastoma phenotypes and niches.

M. Vishnoi, Z. Dereli, Z. Yin, E. K. Kong, M. Kinali, K. Thapa, O. Babur, K. Yun, N. H. Abdelfattah, X. Li, B. Bozorgui, R. C. Rostomily, A. Korkut

This paper identifies a matrix code based on extracellular core matrix proteins that categorizes glioblastoma tumors into prognostic groups and predicts responses to PD1 blockade.

bioRxiv · Jun 6, 2023 · PreprintCancer Biology

BET inhibition induces an anti-apoptotic adaptive response and therapeutic vulnerability to MCL1 inhibitors in breast cancer

G. Yan, H. Wang, A. Luna, B. Bozorgui, X. Li, Z. Dereli, X. Chen, Y. Lu, O. Babur, C. Sander, G. Mills, A. Korkut

This study shows BET inhibition in breast cancer triggers an anti-apoptotic response via MCL1 upregulation, and co-targeting BET and MCL1 is synergistic and effective in basal-like breast cancers.

bioRxiv · Mar 21, 2020Cancer Biology