Author

P. Lazcano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

M. W. Schmidtke· 2 shared C. J. Onu· 1 shared K. C. Case· 1 shared M. Balazova· 1 shared M. Chakkour· 1 shared M. Greenberg· 1 shared M. L. Greenberg· 1 shared M. Ren· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

M. W. Schmidtke

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2 papers

Recent·Most cited

Upregulation of the AMPK-FOXO1-PDK4 pathway is a primary mechanism of pyruvate dehydrogenase activity reduction and leads to increased glucose uptake in tafazzin-deficient cells

Z. Liang, T. Ralph-Epps, M. W. Schmidtke, P. Lazcano, S. W. Denis, M. Balazova, M. Chakkour, S. Hazime, M. Ren, M. Schlame, R. Houtkooper, M. Greenberg

This study identifies the upregulation of the AMPK-FOXO1-PDK4 pathway as the mechanism causing reduced PDH activity and increased glucose uptake in tafazzin-deficient cells.

bioRxiv · Feb 3, 2024 · PreprintCell Biology

Inositol depletion regulates phospholipid metabolism and activates stress signaling in HEK293T cells

M. Suliman, K. C. Case, M. W. Schmidtke, P. Lazcano, C. J. Onu, M. L. Greenberg

This study shows that inositol depletion in HEK293T cells disrupts phospholipid metabolism and activates stress signaling pathways.

bioRxiv · Feb 21, 2022Cell Biology