Author

P. Sarnow

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

K. Somboonwiwat· 2 shared P. Boonchuen· 2 shared T.-C. Chen· 2 shared G. Perez-Vilaro· 1 shared J. Belk· 1 shared J. Carette· 1 shared J. Diez· 1 shared J. E. Carette· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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2 shared papersMicrobiology

K. Somboonwiwat

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4 papers

Recent·Most cited

Virus-derived circular RNAs populate hepatitis C virus-infected cells

Q. Cao, P. Boonchuen, T.-C. Chen, S. Lei, K. Somboonwiwat, P. Sarnow

The paper identifies virus-derived circular RNAs (vcircRNAs) in hepatitis C virus-infected cells, showing they enhance viral RNA abundance and promote translation of novel viral proteins.

bioRxiv · Jul 28, 2023Microbiology

Loquacious Modulates Flaviviral RNA Replication in Mosquito Cells

S. Shivaprasad, K.-F. Weng, Y. S. Ooi, J. Belk, J. E. Carette, R. Flynn, P. Sarnow

The paper identifies Loquacious as a pro-viral host factor in mosquito cells that modulates flaviviral RNA replication and gene amplification.

bioRxiv · Dec 3, 2021Microbiology

Host-derived Circular RNAs Display Proviral Activities in Hepatitis C Virus - Infected Cells

T.-C. Chen, M. Tallo-Parra, S. Kadener, R. Böttcher, G. Perez-Vilaro, P. Boonchuen, K. Somboonwiwat, J. Diez, P. Sarnow

This study identifies host-derived circular RNAs that promote hepatitis C virus replication by modulating viral gene expression and inhibiting antiviral pathways.

bioRxiv · Jan 24, 2020Microbiology

Impact of a Patient-Derived Hepatitis C Viral RNA Genome with a Mutated MicroRNA Binding Site

M. Mata, S. Neben, K. Majzoub, J. Carette, P. Sarnow

The paper shows that a single-nucleotide mutation in HCV RNA increases replication when miR-122 is low but reduces replication when miR-122 is high.

bioRxiv · Nov 13, 2018Microbiology