Author

Patrick R. Halliday

Department of Medicine, University of California, San Francisco, San Francisco, CA 94158, USA

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Beatrice GiniiD· 1 shared Carlos Gomez· 1 shared Collin M. Blakely· 1 shared D. Lucas Kerr· 1 shared Dora Barbosa· 1 shared Lisa Tan· 1 shared Paul Allegakoen· 1 shared Philippe Gui· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Beatrice GiniORCID

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1 paper

Recent·Most cited

CDK4 or CDK6 upregulation induces DNA replication stress and genomic instability to cause EGFR targeted therapy resistance in lung cancer

Beatrice Gini, Philippe Gui, Wei Wu, D. Lucas Kerr, Lisa Tan, Dora Barbosa, Victor Olivas, Paul Allegakoen, Carlos Gomez, Patrick R. Halliday, Sarah Elmes, Veronica Steri, Turja Chakrabarti, Trever G. Bivona, Collin M. Blakely

This study shows that CDK4/6 upregulation causes DNA replication stress and genomic instability, leading to resistance to EGFR inhibitors in lung cancer, and suggests combining CDK4/6 and EGFR inhibitors as a treatment.

bioRxiv · Mar 12, 2024 · PreprintCancer Biology