Author

R. Inturi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

P. Jemth· 3 shared A. K. Overby· 2 shared A. Sayadi· 2 shared C. Benz· 2 shared E. Andersson· 2 shared F. Mihalic· 2 shared L. Simonetti· 2 shared M. Ali· 2 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersBiochemistry

P. Jemth

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Large-scale phage-based screening reveals extensive pan-viral mimicry of host short linear motifs

F. Mihalic, L. Simonetti, G. Giudice, M. Rubin Sander, R. Lindqvist, M. B. A. Peters, C. Benz, E. Kassa, D. Badgujar, R. Inturi, M. Ali, I. Krystkowiak, A. Sayadi, E. Andersson, H. Aronsson, , Y. Ivarsson

The paper uses phage-based screening to identify 1,712 virus-host interactions mimicking host short linear motifs, revealing therapeutic targets for broad-spectrum antivirals.

bioRxiv · Jun 19, 2022 · PreprintBiochemistry

Large scale discovery of coronavirus-host factor protein interaction motifs reveals SARS-CoV-2 specific mechanisms and vulnerabilities

T. Kruse, C. Benz, D. H. Garvanska, R. Lindqvist, F. Mihalic, F. Coscia, R. Inturi, A. Sayadi, L. Simonetti, E. Nilsson, M. Ali, J. Kliche, A. Moliner Morro, A. Mund, E. Andersson, , Y. Ivarsson

The paper identifies coronavirus-host protein interaction motifs, revealing SARS-CoV-2 specific mechanisms and a peptide-based inhibitor that blocks infection.

bioRxiv · Apr 19, 2021Biochemistry

CRISPR/Cas9-based inactivation of human papillomavirus oncogenes E6 and E7 induces senescence in cervical cancer cells

R. Inturi, P. Jemth

This study shows that CRISPR/Cas9 inactivation of HPV18 E6 and E7 in HeLa cells induces senescence, characterized by specific markers and altered p53/p21 and pRb/p21 levels.

bioRxiv · Apr 16, 2020Cancer Biology