Author

Robert B Cameron

University of Chicago

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Brandon FaubertiD· 2 shared Hardik ShahiD· 2 shared Aaron Y. MochizukiiD· 1 shared Alberto DelaidelliiD· 1 shared Alpaslan TasdoganiD· 1 shared Andrew M. DonsoniD· 1 shared Antonio C.T. Cobos· 1 shared Baoli HuiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCancer Biology

Brandon FaubertORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Adenosylhomocysteinase drives epigenetic and metabolic reprogramming in ZFTA-RELA ependymoma

Navneesh Yadav, Olivia Pelcher, Raja Gopal Reddy Mooli, Robert B Cameron, Alberto Delaidelli, Kannan Boosi Narayana Rao, Bhagyalakshmi Manikandan, Senna Visser, Antonio C.T. Cobos, Vishal Parmar, Yuvraj Kayola, Siva Kumar Natarajan, Yash Patel, Jiao Zhang, Hardik Shah, , Kulandaimanuvel Antony Michealraj

This study identifies AHCY as a key driver of epigenetic and metabolic reprogramming in ZFTA-RELA ependymomas, revealing its role in promoting tumor growth through pyrimidine synthesis and asparagine dependency.

bioRxiv · Oct 7, 2026 · PreprintCancer Biology

MCT1 activity defines an aggressive metabolic phenotype and therapeutic target in non-small cell lung cancer

Robert B Cameron, Kristen Erika Shema, Nathan Dubois, Maia G. Clare, Nia G. Hammond, Mayher Kaur, Brianna K. Chang, Elyse G. Schechter, Juliya Hsiang, Zuzanna Marczenia, Marina C. Garassino, Ling Cai, Christine M. Bestvina, Thomas P. Mathews, Alpaslan Tasdogan, , Brandon Faubert

This study shows that MCT1-mediated lactate uptake sustains nucleotide homeostasis in NSCLC, and its inhibition enhances chemotherapy efficacy by exposing a metabolic vulnerability.

bioRxiv · Sep 22, 2026 · PreprintCancer Biology