Author

S. Marin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

C. Foguet· 2 shared J. J. Lozano· 2 shared M. Cascante· 2 shared T. M. Thomson· 2 shared A. Calvet· 1 shared A. de Lucas-Rius· 1 shared A. Suarez-Bonnet· 1 shared B. D. Lopez-Ayllon· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

C. Foguet

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Glutaminase as a metabolic target of choice to counter acquired resistance to Palbociclib by colorectal cancer cells

M. Tarrado-Castellarnau, C. Foguet, J. Tarrago-Celada, M. Palobart, C. Hernandez-Carro, J. Perarnau, E. Zodda, I. H. Polat, S. Marin, A. Suarez-Bonnet, J. J. Lozano, M. Yuneva, T. M. Thomson, M. Cascante

This study shows that combining Palbociclib with Telaglenestat prevents acquired resistance in colorectal cancer cells by counteracting metabolic reprogramming.

bioRxiv · Jan 4, 2024Cancer Biology

Metabolic and mitochondria alterations induced by SARS-CoV-2 accessory proteins ORF3a, ORF9b, ORF9c and ORF10

B. D. Lopez-Ayllon, S. Marin, M. Farinas Fernandez, T. Garcia-Garcia, R. Fernandez-Rodriguez, A. de Lucas-Rius, N. Redondo, L. Mendoza-Garcia, C. Foguet, J. Grigas, A. Calvet, J. M. Villalba, M. J. Rodriguez Gomez, D. Megias, B. Mandracchia, , M. Montoya

This study identifies mitochondrial and metabolic reprogramming induced by SARS-CoV-2 accessory proteins in lung cells, revealing potential therapeutic targets.

bioRxiv · Sep 26, 2023Immunology