Author

E. Zodda

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

M. Cascante· 3 shared T. M. Thomson· 3 shared J. Tarrago-Celada· 2 shared A. Hibot· 1 shared A. Suarez-Bonnet· 1 shared B. D. Lopez-Ayllon· 1 shared C. Benitez-Rodriguez· 1 shared C. Calvo-Gonzalez· 1 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersMicrobiology

M. Cascante

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Induction of the inflammasome by the SARS-CoV-2 accessory protein ORF9b, abrogated by small-molecule ORF9b homodimerization inhibitors

E. Zodda, M. Pons, N. DeMoya-Valenzuela, C. Calvo-Gonzalez, C. Benitez-Rodriguez, B. D. Lopez-Ayllon, A. Hibot, M. Cascante, M. Montoya, M. Pujol, J. Rubio-Martinez, T. M. Thomson

This study shows that SARS-CoV-2's ORF9b protein activates inflammasomes and caspase-1, and small molecules inhibiting its homodimerization reduce inflammation and restore interferon signaling.

bioRxiv · May 31, 2024Microbiology

Glutaminase as a metabolic target of choice to counter acquired resistance to Palbociclib by colorectal cancer cells

M. Tarrado-Castellarnau, C. Foguet, J. Tarrago-Celada, M. Palobart, C. Hernandez-Carro, J. Perarnau, E. Zodda, I. H. Polat, S. Marin, A. Suarez-Bonnet, J. J. Lozano, M. Yuneva, T. M. Thomson, M. Cascante

This study shows that combining Palbociclib with Telaglenestat prevents acquired resistance in colorectal cancer cells by counteracting metabolic reprogramming.

bioRxiv · Jan 4, 2024Cancer Biology

Cell-autonomous metabolic reprogramming and oxidative stress underlie endothelial dysfunction in acute myocardial infarction

E. Zodda, O. Tura-Ceide, N. L. Mills, J. Tarrago-Celada, M. Carini, T. M. Thomson, M. Cascante

This study identifies metabolic reprogramming and oxidative stress in endothelial cells from acute myocardial infarction patients as drivers of endothelial dysfunction.

bioRxiv · Feb 28, 2023Cell Biology