Author

S. Marwitz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

T. Goldmann· 3 shared A. Gross· 1 shared A. Hoelscher· 1 shared A. Kispert· 1 shared A. Korfiati· 1 shared A. Marazioti· 1 shared B. Kalsdorf· 1 shared C. Hoelscher· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersCancer Biology

T. Goldmann

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Author profile →

3 papers

Recent·Most cited

The pregnancy-associated protein glycodelin as a potential sex-specific target for resistance to immunotherapy in non-small cell lung cancer

S. Richtmann, S. Marwitz, T. Muley, H. Koistinnen, P. Christopoulos, M. Thomas, D. N. Kazdal, M. Allgaeuer, H. Winter, T. Goldmann, M. Meister, U. Klingmueller, M. A. Schneider

This paper identifies glycodelin as a sex-specific factor influencing immunotherapy resistance in NSCLC, showing it impairs survival in female patients and interacts with immune cells.

bioRxiv · Mar 2, 2023Cancer Biology

Immune-evasion of KRAS-mutant lung adenocarcinoma mediated by cAMP response element-binding protein

G. A. Giotopoulou, G. Ntaliarda, A. Marazioti, I. Lilis, F. Kalogianni, E. Tourkochristou, N. Spiropoulou, I. Giopanou, M. Spella, M. Iliopoulou, A. Korfiati, T. Mantamadiotis, C. Rosero, T. Goldmann, S. Marwitz, G. T. Stathopoulos

This study identifies CREB-mediated immune evasion in KRAS-mutant lung adenocarcinoma by suppressing neutrophil recruitment through CXCR1 signaling.

bioRxiv · Jun 19, 2021 · PreprintCancer Biology

WNT6-ACC2-induced accumulation of triacylglycerol rich lipid droplets is exploited by Mycobacterium tuberculosis

J. Brandenburg, S. Marwitz, S. C. Tazoll, F. Waldow, B. Kalsdorf, T. Vierbuchen, T. Scholzen, A. Gross, S. Goldenbaum, A. Hoelscher, M. Hein, L. Linnemann, M. Reimann, A. Kispert, M. Leitges, , N. Reiling

This study shows that the WNT6-ACC2 pathway promotes lipid droplet accumulation in macrophages, which Mycobacterium tuberculosis exploits for growth, and that inhibiting this pathway enhances anti-tuberculosis treatment.

bioRxiv · Jun 26, 2020 · PreprintImmunology