Author

P. Christopoulos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

M. A. Schneider· 2 shared M. Thomas· 2 shared T. Muley· 2 shared A. Corthay· 1 shared A. Schulze· 1 shared A.-K. Daum· 1 shared C. W. Wold· 1 shared D. Dzovor· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersCancer Biology

M. A. Schneider

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer-associated fibroblasts promote drug resistance in ALK-driven lung adenocarcinoma cells by upregulating lipid biosynthesis

A.-K. Daum, L. Schlicker, M. A. Schneider, T. Muley, U. Klingmüller, A. Schulze, M. Thomas, P. Christopoulos, H. Sültmann

This study shows cancer-associated fibroblasts enhance drug resistance in ALK-driven lung adenocarcinoma by promoting lipid biosynthesis and suppressing lipid peroxidation through AKT signaling.

bioRxiv · Aug 8, 2023 · PreprintCancer Biology

The pregnancy-associated protein glycodelin as a potential sex-specific target for resistance to immunotherapy in non-small cell lung cancer

S. Richtmann, S. Marwitz, T. Muley, H. Koistinnen, P. Christopoulos, M. Thomas, D. N. Kazdal, M. Allgaeuer, H. Winter, T. Goldmann, M. Meister, U. Klingmueller, M. A. Schneider

This paper identifies glycodelin as a sex-specific factor influencing immunotherapy resistance in NSCLC, showing it impairs survival in female patients and interacts with immune cells.

bioRxiv · Mar 2, 2023Cancer Biology

Polysaccharides from the fungus Inonotus obliquus activate macrophages into a tumoricidal phenotype via interaction with TLR2, TLR4 and Dectin-1a

C. W. Wold, P. Christopoulos, M. Arias, D. Dzovor, I. Oynebraaten, A. Corthay, K. T. Inngjerdingen

This study shows that acidic polysaccharides from Inonotus obliquus activate macrophages into a tumoricidal phenotype through interactions with TLR2, TLR4, and Dectin-1a.

bioRxiv · Nov 11, 2020 · PreprintImmunology