Author

S. Mendonca

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Microbiology· 2 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 6 Research Focus:cell type annotation· 3 Research Focus:host-pathogen & immune response· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:proteomics· 2 Organism:Arabidopsis· 1

Frequent Co-Authors

F. Martins· 2 shared M. J. Oliveira· 2 shared S. Velho· 2 shared A. Freitas· 1 shared A. G. Ferreira· 1 shared A. L. Machado· 1 shared A. Ribeiro· 1 shared A. Sporbert· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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2 shared papersCancer Biology

F. Martins

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

High capacity of Brazilian Aedes aegypti populations to transmit a locally circulating lineage of Chikungunya virus

A. Freitas, F. Rezende, S. Mendonca, L. Baldon, E. Silva, F. Ferreira, J. Almeida, S. Amadou, B. Marcal, S. Grangeiro, M. Rocha, H. Fritsch, M. Giovanetti, L. Alcantara, L. Moreira, A. G. Ferreira

This study shows Brazilian Aedes aegypti populations efficiently transmit a locally circulating Chikungunya virus ECSA lineage, contributing to its high prevalence.

bioRxiv · Oct 23, 2023 · PreprintImmunology

Modulation of fibroblasts phenotype by colorectal cancer cells-secreted factors is mostly independent of oncogenic KRAS

P. Dias Carvalho, S. Mendonca, F. Martins, M. J. Oliveira, S. Velho

This study shows that mutant KRAS in colorectal cancer cells has a limited role in modulating fibroblast pro-tumorigenic features, suggesting KRAS-independent mechanisms may drive fibroblast activation and resistance to KRAS inhibitors.

Cells · Aug 11, 2022Peer-reviewedCancer Biology

Mutant KRAS modulates colorectal cancer cells invasive response to fibroblast-secreted factors through the HGF/C-MET axis

P. Dias-Carvalho, F. Martins, S. Mendonca, A. Ribeiro, A. L. Machado, J. Carvalho, M. J. Oliveira, S. Velho

This study shows mutant KRAS modulates colorectal cancer cell invasion via the HGF/C-MET axis, influenced by fibroblast-secreted factors.

bioRxiv · Nov 16, 2021Cancer Biology

Putative Pseudolysogeny-Dependent Phage Gene Implicated in the Superinfection Resistance of Cutibacterium acnes

S. Wottrich, S. Mendonca, C. Safarpour, C. Nguyen, L. J. Marinelli, R. L. Modlin, J. Moberg Parker

The paper identifies a phage gene involved in superinfection resistance of C. acnes through pseudolysogeny.

bioRxiv · Dec 15, 2020Microbiology

IFT88 transports Gucy2d, a guanylyl cyclase, to maintain sensory cilia function in Drosophila

S. Werner, S. Zitouni, P. Okenve-Ramos, S. Mendonca, A. Sporbert, C. Spalthoff, M. C. Gopfert, S. C. Jana, M. Bettencourt-Dias

This study identifies IFT88 as a transporter of Gucy2d to sensory cilia in Drosophila, showing its role in maintaining ciliary function and sensory behavior.

bioRxiv · Dec 15, 2020 · PreprintCell Biology

Herpes Simplex Virus Infection, Acyclovir and IVIG Treatment All Independently Cause Gut Dysbiosis.

E. M. Cantin, C. Ramakrishna, S. Mendonca, P. Reugger, J. H. Kim, J. Borneman

This study shows HSV, ACV, and IVIG independently cause sex-biased gut dysbiosis in mice, with ACV reducing protective bacterial taxa linked to GVHD prevention in HSCT patients.

bioRxiv · Nov 20, 2019Microbiology