Author

Stephen J. Morris

Therapure Biopharma (Canada)

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Alejandro Álvarez-QuilóniD· 2 shared Artur VelosoiD· 2 shared Jordan T.F. Young· 2 shared Julian Bowlan· 2 shared Li Li· 2 shared Michal ZimmermanniD· 2 shared Nancy LaterreuriD· 2 shared Abira Rajah· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersCancer Biology

Alejandro Álvarez-QuilónORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

CRISPR-enabled genetic screens identify synthetic lethal targets across frequently altered cancer drivers

Jessica Desjardins, Julian Bowlan, Cynthia Bernier, Srishti Jain, Théo Goullet de Rugy, David Gallo, Mark E. Orcholski, Nancy Laterreur, Abira Rajah, Joshua Miller, Jennifer A. Lafontaine, Vivek Bhaskaran, Li Li, Alexanda K. Ling, Jesse H. Leblanc, , Jordan T.F. Young

This study identifies synthetic lethal interactions between cancer drivers and tractable targets using CRISPR screens and cancer dependency data, offering new drug targets for therapy.

bioRxiv · Jan 25, 2026 · PreprintCancer Biology

A dual mechanism of sensitivity to PLK4 inhibition by RP-1664 in neuroblastoma

Isabel Soria‐Bretones, Matias Casás‐Selves, Elliot Goodfellow, Li Li, Cathy Caron, Ariya Shiwram, Hye‐Yeon Kim, Danielle Henry, Nancy Laterreur, Julian Bowlan, Alejandro Álvarez-Quilón, Frédéric Vallée, Artur Veloso, Jordan T.F. Young, Marc L. Hyer, , Michal Zimmermann

This study shows neuroblastoma cells are hypersensitive to PLK4 inhibitor RP-1664 due to dual mechanisms of centriole loss and amplification, leading to cell death.

bioRxiv · Feb 15, 2025 · PreprintCancer Biology