Author

T. Harm

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Bayrak· 1 shared A.-K. Rohlfing· 1 shared D. Schaale· 1 shared K. A. L. Mueller· 1 shared M. Gawaz· 1 shared M. Sigle· 1 shared R. Hochuli· 1 shared S. Laufer· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Bayrak

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1 paper

Recent·Most cited

Hemin-induced platelet activation is regulated via ACKR3 chemokine surface receptor - implications for passivation of vulnerable atherosclerotic plaque

Z. Laspa, V. Dicenta-Baunach, D. Schaale, M. Sigle, R. Hochuli, T. Castor, A. Bayrak, T. Harm, K. A. L. Mueller, T. Pillaiyar, S. Laufer, A.-K. Rohlfing, M. Gawaz

This study shows hemin activates platelets and promotes thrombosis, which can be inhibited by ACKR3 agonists, suggesting a potential therapeutic strategy for atherosclerotic plaque vulnerability.

bioRxiv · May 13, 2024 · PreprintCell Biology