Author

Thomas O. Moninger

University of Iowa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Genetic & Genomic Medicine· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell-cell communication· 1 Assay:imaging· 1 Organism:plant (other)· 1 Assay:proteomics· 1

Frequent Co-Authors

Alejandro A. PezzuloiD· 2 shared David Kyle MeyerholziD· 2 shared Douglas B. HornickiD· 2 shared Akansha JainiD· 1 shared Amy L. RyaniD· 1 shared Anthony J. FischeriD· 1 shared Beck E. Fitzpatrick· 1 shared Benjamin W. DarbroiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

Alejandro A. PezzuloORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Defining Functional Correction Thresholds in Primary Ciliary Dyskinesia for Effective Gene Therapies

Beck E. Fitzpatrick, Brett J. Wineinger, Jason E. Babcock, Erik J. Quiroz, Emily C. Liu, Lalit Kumar Gautam, Alejandro A. Pezzulo, Thomas O. Moninger, David Kyle Meyerholz, Douglas B. Hornick, Amy L. Ryan

This study defines a functional correction threshold for gene therapies in PCD by analyzing how mixed populations of mutant and wild-type ciliated cells affect mucociliary transport.

bioRxiv · Feb 20, 2026 · PreprintCell Biology

Heterozygous KRT32 variant is responsible for autosomal dominant loose anagen hair syndrome

Marcelo Melo, Elizabeth S. Phillippi, Thomas O. Moninger, Kya Foxx, Benjamin W. Darbro, Kelly N. Messingham, Edward A. Sander, Hatem El‐Shanti

The paper identifies a heterozygous KRT32 variant causing autosomal dominant loose anagen hair syndrome and shows it reduces binding affinity to KRT82, impairing hair shaft anchoring.

medRxiv · May 2, 2025 · PreprintGenetic & Genomic Medicine

Development and Initial Characterization of Pigs with DNAI1 Mutations and Primary Ciliary Dyskinesia

Mahmoud H. Abou Alaiwa, Brieanna M. Hilkin, Margaret P. Price, Nicholas D. Gansemer, Michael R. Rector, Mal R. Stroik, Linda S. Powers, Kristin M. Whitworth, Melissa Samuel, Akansha Jain, Lynda S. Ostedgaard, Sarah E. Ernst, Winter Philibert, Linda D. Boyken, Thomas O. Moninger, , David Anthony Stoltz

This paper develops a PCD pig model with DNAI1 mutations and characterizes its respiratory and sinus disease features, showing similarities to human PCD.

bioRxiv · May 22, 2024 · PreprintPhysiology