Epileptic phenotypes in slc13a5 loss-of-function zebrafish are rescued by blocking NMDA receptor signaling
Deepika Dogra, Van Anh Phan, Cezar Gavrilovici, Nadia DiMarzo, Kingsley Ibhazehiebo, Deborah Marie Kurrasch
This study shows that loss-of-function mutations in zebrafish slc13a5 cause epilepsy-like phenotypes, which are rescued by blocking NMDA receptor signaling.
bioRxiv · Jan 15, 2024 · PreprintNeuroscience