Author

Zerina Balic

Icahn School of Medicine at Mount Sinai

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:database / resource· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Dirk HubmacheriD· 2 shared Alexander S. OkamotoiD· 1 shared Alice H. HuangiD· 1 shared Belinda B. WillardiD· 1 shared Daniel J. RichardiD· 1 shared Daniel Martin· 1 shared Daniel Medina-CanoiD· 1 shared Deepak A. KajiiD· 1 shared

Activity by Year

2020-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCell Biology

Dirk HubmacherORCID

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Author profile →

3 papers

Recent·Most cited

A mouse organoid platform for modeling cerebral cortex development and cis-regulatory evolution in vitro

Daniel Medina-Cano, Mohammed Tarek Islam, Veronika Petrova, Sanjana Dixit, Zerina Balic, Marty G. Yang, Matthias Stadtfeld, Emily S. W. Wong, Thomas Vierbuchen

The paper develops a mouse cortical organoid platform to study cis-regulatory evolution and identifies genes with dynamic allelic imbalances during neurogenesis.

Developmental Cell · Aug 27, 2025Peer-reviewedDevelopmental Biology

Combined ADAMTS10 and ADAMTS17 inactivation exacerbates bone shortening and skin phenotypes

Nandaraj Taye, Stylianos Z. Karoulias, Zerina Balic, Lauren Wang, Belinda B. Willard, Daniel Martin, Daniel J. Richard, Alexander S. Okamoto, Terence Dante Capellini, Suneel Apte, Dirk Hubmacher

This study shows that inactivating both ADAMTS10 and ADAMTS17 in mice causes severe bone shortening and skin issues, suggesting they regulate ECM formation and chondrocyte function.

bioRxiv · Jan 23, 2025 · PreprintCell Biology

TGFβ signaling is required for tenocyte recruitment and functional neonatal tendon regeneration

Deepak A. Kaji, Kristen Howell, Zerina Balic, Dirk Hubmacher, Alice H. Huang

This study identifies TGFβ signaling as critical for neonatal tendon regeneration by promoting tenocyte recruitment and functional recovery.

eLife · Jun 5, 2020Peer-reviewedDevelopmental Biology