Author

A. Arner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Assay:electrophysiology· 1

Frequent Co-Authors

A. Kahsay· 1 shared A. Silvani· 1 shared B. Fehse· 1 shared C. Berteotti· 1 shared C. D. Baldus· 1 shared C. Miehting· 1 shared D. M. Schewe· 1 shared F. Pedrosa Domellof· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCell Biology

A. Kahsay

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Author profile →

3 papers

Recent·Most cited

fhl2b expression ameliorates muscular dystrophy

N. Dennhag, A. Kahsay, I. Nissen, M. Chermenina, H. Nord, J. Liu, A. Arner, J.-X. Liu, L. J. Backman, S. Remeseiro, J. von Hofsten, F. Pedrosa Domellof

This study identifies fhl2b as a protective gene that ameliorates muscular dystrophy in zebrafish by rescuing muscle phenotype and improving survival.

bioRxiv · Oct 26, 2023 · PreprintCell Biology

In vivo inducible reverse genetics in patients' tumors to identify individual therapeutic targets

M. Carlet, K. Voelse, J. Vergalli, M. Becker, T. Herold, A. Arner, W.-H. Liu, V. Dill, B. Fehse, C. D. Baldus, L. Bastian, L. Lenk, D. M. Schewe, J. W. Bagnoli, P. J. Jost, , I. Jeremias

The paper develops an in vivo inducible RNAi system in patient-derived xenograft models to identify therapeutic targets in acute leukemias.

bioRxiv · May 2, 2020Cancer Biology

Long-term cardiovascular re-programming by short-term perinatal exposure to nicotine‘s main metabolite cotinine

S. Bastianini, V. Lo Martire, A. Silvani, G. Zoccoli, C. Berteotti, H. Lagercrantz, A. Arner, G. Cohen

This study shows that perinatal exposure to cotinine, a nicotine metabolite, causes long-term cardiovascular changes in mice, suggesting potential developmental risks.

bioRxiv · Sep 23, 2017Physiology