Author

A. Dahmani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

E. Marangoni· 3 shared E. Montaudon· 2 shared A. Durand· 1 shared A. Eisele· 1 shared A. Trouchet· 1 shared A.-M. Lyne· 1 shared A.-S. Mace· 1 shared C. Colombero· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersCancer Biology

E. Marangoni

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Author profile →

3 papers

Recent·Most cited

mTOR repression in response to amino acid starvation promotes ECM degradation through MT1-MMP endocytosis arrest

C. Colombero, D. Remy, S. Antoine, A.-S. Mace, P. Monteiro, N. ElKhatib, M. Fournier, A. Dahmani, E. Montaudon, G. Montagnac, E. Marangoni, P. Chavrier

The paper shows that mTOR repression during amino acid starvation leads to increased ECM degradation via MT1-MMP endocytosis arrest in tumor cells.

bioRxiv · Jan 29, 2021Cancer Biology

H3K27me3 is a determinant of chemotolerance in triple-negative breast cancer

J. Marsolier, P. Prompsy, A. Durand, A.-M. Lyne, C. Landragin, A. Trouchet, S. Tenreira-bento, A. Eisele, S. Foulon, L. Baudre, K. Grosselin, M. Bohec, S. Baulande, A. Dahmani, E. Letouze, , C. Vallot

This study identifies H3K27me3 as a key determinant of chemotolerance in triple-negative breast cancer by analyzing single-cell epigenomic and transcriptomic data.

bioRxiv · Jan 4, 2021 · PreprintCancer Biology

GATA3 and MDM2 are synthetic lethal in estrogen receptor-positive breast cancers

G. Bianco, M. Coto Llerena, J. Gallon, S. Taha-Mehlitz, V. Kancherla, M. Konantz, S. Srivatsa, H. Montazeri, F. Panebianco, M. De Menna, V. Paradiso, C. Ercan, A. Dahmani, E. Montaudon, N. Beerenwinkel, , S. Piscuoglio

This study identifies GATA3 and MDM2 as synthetic lethal in ER-positive breast cancers, showing MDM2 inhibition can target GATA3-deficient tumors via the PI3K/Akt/mTOR pathway.

bioRxiv · May 18, 2020Cancer Biology