Author

A. Halama

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:metabolomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:metabolism & bioenergetics· 1 Approach:statistical model· 1 Organism:Xenopus· 1

Frequent Co-Authors

K. Suhre· 3 shared S. S. Gross· 2 shared A. Iskandarani· 1 shared A. M. Bhagwat· 1 shared A. Rafii· 1 shared G. Kastenmueller· 1 shared G. Thareja· 1 shared I. W. Achka· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersCancer Biology

K. Suhre

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Progesterone induces meiosis through two obligate co-receptors with PLA2 activity

N. Nader, L. Assaf, L. Zarif, A. Halama, S. Yadav, M. Dib, N. Attarwala, Q. Chen, K. Suhre, S. S. Gross, K. Machaca

This study identifies ABHD2 as an essential co-receptor for mPRβ to trigger meiosis via PLA2 activity in Xenopus oocytes.

bioRxiv · Sep 9, 2023Cell Biology

Metabolic dysregulations of cancer cells with metastatic potential

S. Abdul Kader, S. Dib, I. W. Achka, G. Thareja, K. Suhre, A. Rafii, A. Halama

This study identifies metabolic differences between high and low metastatic breast cancer cell lines, highlighting key metabolic pathways and molecules involved in metastasis.

bioRxiv · Jun 2, 2021 · PreprintCancer Biology

Accelerated lipid catabolism and autophagy are cancer survival mechanisms under inhibited glutaminolysis

A. Halama, M. Kulinski, S. S. Dib, S. B. Zaghlool, K. S. Siveen, A. Iskandarani, N. J. Satheesh, A. M. Bhagwat, S. Uddin, G. Kastenmueller, O. Elemento, S. S. Gross, K. Suhre

The paper shows that inhibiting glutaminolysis in cancer cells leads to lipid catabolism and autophagy, which promote survival, and that combined inhibition of both pathways can induce cell death.

bioRxiv · Jan 15, 2018Cancer Biology