Author

A. Parini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

A. Henras· 1 shared A. Lagnado· 1 shared A. Walaszczyk· 1 shared A.-C. Godet· 1 shared A.-C. Prats· 1 shared B. Garmy-Susini· 1 shared C. Correia-Melo· 1 shared C. Proctor· 1 shared

Activity by Year

2018-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Henras

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Vasohibin1, a new IRES trans-acting factor for induction of (lymph)angiogenic factors in early hypoxia

F. Hantelys, A.-C. Godet, F. David, F. Tatin, E. Renaud-Gabardos, F. Pujol, L. Diallo, I. Ader, L. Ligat, A. Henras, Y. Sato, E. Lacazette, A. Parini, B. Garmy-Susini, A.-C. Prats

The paper identifies vasohibin1 as an IRES trans-acting factor that activates angiogenic IRESs in early hypoxia while inhibiting others in normoxia.

bioRxiv · Jul 11, 2019 · PreprintCell Biology

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani, A. Walaszczyk, E. Dookun, J. Chapman, J. Birch, H. Salmonowicz, M. Ogrodnik, D. Jurk, C. Proctor, C. Correia-Melo, S. Victorelli, E. Fielder, R. Berlinguer-Palmini, , J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

bioRxiv · Aug 17, 2018Cell Biology