Author

M. Ogrodnik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1 Scientific Communication & Education· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Lagnado· 1 shared A. Parini· 1 shared A. Walaszczyk· 1 shared B. Bachmann· 1 shared B. Fenesi· 1 shared B. Schaedl· 1 shared C. Correia-Melo· 1 shared C. Proctor· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Lagnado

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The p-rpS6-zone delineates wounding response and the healing process

N. A. R. Ring, H. Dworak, B. Bachmann, B. Schaedl, K. Valdivieso, T. Rozmaric, P. Heimel, K. Schuetzenberger, G. Leinfellner, J. Ferguson, S. Drechsler, M. Mildner, M. Schosserer, P. Slezak, O. Meyuhas, , M. Ogrodnik

The paper identifies a p-rpS6-zone around wounds that marks the healing process and shows its role in tissue repair.

bioRxiv · Sep 27, 2022 · PreprintPhysiology

A Mental Health Paradox: Mental health was both a motivator and barrier to physical activity during the COVID-19 pandemic

M. Y. Marashi, E. Nicholson, M. Ogrodnik, B. Fenesi, J. J. Heisz

This study examines how mental health influenced physical activity during the pandemic, finding that deteriorated mental health reduced activity while mental health benefits motivated some to stay active.

bioRxiv · Sep 3, 2020Scientific Communication & Education

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani, A. Walaszczyk, E. Dookun, J. Chapman, J. Birch, H. Salmonowicz, M. Ogrodnik, D. Jurk, C. Proctor, C. Correia-Melo, S. Victorelli, E. Fielder, R. Berlinguer-Palmini, , J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

bioRxiv · Aug 17, 2018Cell Biology