Author

A. Sol

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Organism:chicken / avian· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

E. Lipo· 2 shared R. R. Isberg· 2 shared A. Rouvinski· 1 shared C. Murphy· 1 shared C. S. Fraser· 1 shared D. deJesus-Diaz· 1 shared M. Devereux· 1 shared M. Kumar· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersMicrobiology

E. Lipo

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Primary bovine white blood cells support dissemination of Lumpy Skin Disease Virus while suppressing viral replication

M. Kumar, O. Frid, A. Sol, A. Rouvinski, S. Karniely

This study shows that primary bovine white blood cells support LSDV dissemination but suppress viral replication, with heat-treated virus inducing antiviral gene expression.

bioRxiv · Jul 18, 2024 · PreprintMicrobiology

5' Untranslated mRNA Regions Allow Bypass of Host Cell Translation Inhibition by Legionella pneumophila

E. Lipo, S. Asrat, W. Huo, A. Sol, C. S. Fraser, R. R. Isberg

This study identifies host proteins selectively synthesized during Legionella pneumophila infection and shows that 5' untranslated regions (5UTRs) enable translation of low-abundance transcripts despite host translation inhibition.

bioRxiv · Apr 29, 2022Microbiology

Legionella pneumophila translocated translation inhibitors are required for bacterial-induced host cell cycle arrest

A. Sol, D. deJesus-Diaz, C. Murphy, M. Devereux, E. Lipo, R. R. Isberg

This study shows that Legionella pneumophila uses translation inhibitors to block host cell cycle progression and prevent entry into S phase, which is toxic for bacterial replication.

bioRxiv · Nov 28, 2018Microbiology