Author

C. Daunt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. Marsland· 2 shared G. Coakley· 2 shared m. Moyat· 2 shared N. Harris· 2 shared A. A. T. Smith· 1 shared A. Butler· 1 shared A. Zaini· 1 shared B. Volpe· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

B. Marsland

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2 papers

Recent·Most cited

Holding glycolysis in check though Alox15 activity is required for macrophage M2 commitment and function in tissue repair and anti-helminth immunity.

R. Doolan, m. Moyat, G. Coakley, L. Wickramasinghe Mudiyansel, C. Daunt, B. Volpe, F. Henkel, V. Trefzer, N. Ubags, A. Butler, R. Chatzis, B. Marsland, A. A. T. Smith, D. L. Deveson, E. N. S. McGowan, , N. Harris

bioRxiv · Mar 26, 2024 · PreprintImmunology

Helminth infection driven gastrointestinal hypermotility is independent of eosinophils and mediated by alterations in smooth muscle instead of enteric neurons

N. Harris, K. Barry, G. Coakley, M. Moyat, C. Daunt, A. Zaini, L. Wickramasinghe, B. Yumnam, M. Camberis, G. Le Gros, O. Perdijk, J. Foong, J. Bornstein, B. Marsland

This study shows that helminth-induced intestinal hypermotility in mice is driven by smooth muscle changes, not eosinophils or the enteric nervous system.

bioRxiv · Oct 23, 2023Immunology