Author

C. Deisl

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Assay:imaging· 2 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

D. W. Hilgemann· 2 shared D. G. Fuster· 1 shared D. Konrad· 1 shared G. Albano· 1 shared J. Chung· 1 shared O. W. Moe· 1 shared P. Kucharczyk· 1 shared S. Wueest· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 12 mutual co-author links

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2 shared papersCell Biology

D. W. Hilgemann

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Author profile →

3 papers

Recent·Most cited

Constitutive Plasma Membrane Turnover in T-REx293 cells via Ordered Membrane Domain Endocytosis under Mitochondrial Control

C. Deisl, O. W. Moe, D. W. Hilgemann

This study identifies ordered membrane domain endocytosis (OMDE) as a constitutive, G-protein-dependent endocytic mechanism in T-REx293 cells, regulated by mitochondrial metabolism and palmitoylation.

bioRxiv · Jan 17, 2024 · PreprintCell Biology

Pore-like diffusion barriers in murine cardiac myocytes

C. Deisl, J. Chung, D. W. Hilgemann

The paper measures solute diffusion in cardiac myocytes and finds that diffusion is significantly restricted, likely due to myofilament and mitochondrial networks, with porin channels allowing faster diffusion.

bioRxiv · Jan 2, 2023 · PreprintPhysiology

Thiazides induce glucose intolerance through inhibition of mitochondrial carbonic anhydrase 5b in β-cells

P. Kucharczyk, G. Albano, C. Deisl, S. Wueest, D. Konrad, D. G. Fuster

This study identifies mitochondrial carbonic anhydrase 5b (CA5b) as the molecular target of thiazides, showing its inhibition leads to reduced insulin secretion and glucose intolerance in β-cells.

bioRxiv · Mar 23, 2022 · PreprintPhysiology