Author

C. Krieg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

S. Guglietta· 3 shared A. C. Koong· 1 shared A. Easton· 1 shared A. J. Giaccia· 1 shared B. Fosso· 1 shared C. Beach· 1 shared D. Jiang· 1 shared D. K. Nambiar· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

S. Guglietta

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Innate immune receptor C5aR1 regulates cancer cell fate and can be targeted to improve radiotherapy in tumours with immunosuppressive microenvironments

C. Beach, D. MacLean, D. Majorova, S. Melemenidis, D. K. Nambiar, R. K. Kim, G. N. Valbuena, S. Guglietta, C. Krieg, M. D. Damavandi, T. Suwa, A. Easton, E. Domingo, E. J. Moon, D. Jiang, , M. M. Olcina

This study identifies C5aR1 as a targetable receptor on cancer cells that enhances radiotherapy efficacy in immunosuppressive tumours by promoting apoptosis without affecting normal tissues.

bioRxiv · Jan 10, 2023 · PreprintCancer Biology

Use of CD122+ natural killer cell precursors for superior anti-leukemia responses.

S. Guglietta, L. Cardenas, C. Krieg

This study shows that IL-2cx treatment generates and expands NK cells that effectively control AML in vivo without requiring adoptive transfer.

bioRxiv · Oct 24, 2022 · PreprintImmunology

Loss of C3aR induces immune infiltration and inflammatory microbiota in a novel spontaneous model of colon cancer

C. Krieg, L. M. Weber, B. Fosso, G. Hardiman, E. Mileti, S. El Aidy, M. Marzano, M. D. Robinson, S. Guglietta

This study shows that loss of C3aR in a mouse model of colon cancer leads to immune infiltration and inflammatory microbiota changes.

bioRxiv · Jan 18, 2021 · PreprintImmunology