TRIM9-dependent ubiquitination of DCC constrains kinase signaling, exocytosis, and axon branching
M. Plooster, S. Menon, C. C. Winkle, F. L. Urbina, C. Monkiewicz, K. D. Phend, R. J. Weinberg, S. L. Gupton
This study shows TRIM9 ubiquitinates DCC, inhibiting FAK activation, which regulates exocytosis and axon branching in neurons.
bioRxiv · Jun 23, 2017Cell Biology