Author

D. K. Dang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Research Focus:cancer & tumor microenvironment· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

R. Rao· 4 shared M. Ko· 3 shared M. R. Makena· 3 shared H. Prasad· 2 shared A. X. Mekile· 1 shared C. Talbot· 1 shared J. M. Warrington· 1 shared J. P. Llongueras· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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4 shared papersCancer Biology

R. Rao

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Epigenetic Modulation of SPCA2 Reverses Epithelial to Mesenchymal Transition in Breast Cancer Cells

M. R. Makena, M. Ko, D. K. Dang, R. Rao

This study shows that histone deacetylase inhibitors increase SPCA2 levels, activate Ca2+ signaling, and reverse epithelial to mesenchymal transition in triple receptor negative breast cancer cells.

bioRxiv · Nov 12, 2020Cancer Biology

Store Independent Ca2+ Entry Regulates the DNA Damage Response in Breast Cancer Cells

M. R. Makena, M. Ko, A. X. Mekile, D. K. Dang, J. M. Warrington, P. J. Buckhaults, C. Talbot, R. Rao

This study identifies SPCA2 as a driver of chemotherapy resistance in ER+/p53WT breast cancer by suppressing the DNA damage response through store-independent Ca2+ entry.

bioRxiv · Apr 6, 2020 · PreprintCancer Biology

A Ca2+-ATPase Regulates E-cadherin Biogenesis and Epithelial-Mesenchymal Transition in Breast Cancer Cells

D. K. Dang, M. R. Makena, J. P. Llongueras, H. Prasad, M. Ko, M. Bandral, R. Rao

This study identifies SPCA2 as a regulator of E-cadherin biogenesis and epithelial-mesenchymal transition in breast cancer, independent of its Ca2+ pump activity.

bioRxiv · Mar 17, 2019Cancer Biology

NHA2 promotes cyst development in an in vitro model of polycystic kidney disease

H. Prasad, D. K. Dang, K. C. Kondapalli, N. Natarajan, V. Cebotaru, R. Rao

This study identifies NHA2 as a regulator of cyst development in ADPKD by showing its up-regulation correlates with disease severity and its causal role in increased cyst size in vitro.

bioRxiv · Jul 7, 2018Physiology