Author

D. M. Renner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Assay:bulk RNA-seq· 1 Assay:proteomics· 1

Frequent Co-Authors

S. R. Weiss· 3 shared A. Fausto· 1 shared A. Gonzalez· 1 shared A. Mohamed· 1 shared A. Valdespino· 1 shared B. Dong· 1 shared C. Dann· 1 shared C. J. Otter· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersMicrobiology

S. R. Weiss

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3 papers

Recent·Most cited

Interferon signaling in the nasal epithelium distinguishes among lethal and common cold respiratory viruses and is critical for viral clearance

C. J. Otter, D. M. Renner, A. Fausto, L. H. Tan, N. A. Cohen, S. R. Weiss

The paper examines how interferon signaling in nasal epithelial cells differentiates between common cold and lethal respiratory viruses, showing its role in viral clearance.

bioRxiv · Dec 18, 2023 · PreprintMicrobiology

SARS-CoV-2 diverges from other betacoronaviruses in only partially activating the IRE1α/XBP1 ER stress pathway in human lung-derived cells

L. C. Nguyen, D. M. Renner, D. Silva, D. Yang, N. Parenti, K. M. Medina, V. Nicolaescu, H. Gula, N. Drayman, A. Valdespino, A. Mohamed, C. Dann, K. Wannemo, L. Robinson-Mailman, A. Gonzalez, , S. R. Weiss

This study compares how SARS-CoV-2 and other betacoronaviruses activate the IRE1α/XBP1 ER stress pathway in human lung cells, finding that SARS-CoV-2 only partially activates it, potentially evading host immune detection.

bioRxiv · Dec 30, 2021 · PreprintMicrobiology

The host antiviral ribonuclease L protein supports Zika virus replication factory formation to enhance infectious virus production

J. N. Whelan, J. Hatterschide, D. M. Renner, B. Dong, R. H. Silverman, S. R. Weiss

This study shows RNase L supports ZIKV replication factory formation, enhancing infectious virus production, but inhibits other flaviviruses like dengue and West Nile.

bioRxiv · Nov 24, 2019 · PreprintMicrobiology