Author

D. Miroshnychenko

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Marusyk· 2 shared D. Basanta· 2 shared P. M. Altrock· 2 shared A. C. Tan· 1 shared A. K. Miller· 1 shared E. Baratchart· 1 shared M. A. Laurie· 1 shared M. Bui· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

A. Marusyk

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2 papers

Recent·Most cited

Paracrine enhancement of tumor cell proliferation provides indirect stroma-mediated chemoresistance via acceleration of tumor recovery between chemotherapy cycles.

D. Miroshnychenko, T. Miti, P. Kumar, A. K. Miller, M. Laurie, N. Giraldo, M. M. Bui, P. M. Altrock, D. Basanta, A. Marusyk

The paper investigates how stromal factors indirectly enhance tumor chemoresistance by accelerating tumor recovery between chemotherapy cycles through paracrine signaling.

bioRxiv · Feb 7, 2023 · PreprintCancer Biology

Spontaneous Cell Fusions as a Mechanism of Parasexual Recombination in Tumor Cell Populations

D. Miroshnychenko, E. Baratchart, M. Ferrall-Fairbanks, R. Vander Velde, M. A. Laurie, M. Bui, A. C. Tan, P. M. Altrock, D. Basanta, A. Marusyk

This study investigates spontaneous cell fusions in tumor cell populations as a mechanism of parasexual recombination, finding evidence of low-frequency fusions and their role in increasing clonal heterogeneity.

bioRxiv · Mar 9, 2020Cancer Biology