Author

D. Vetrie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:clustering· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Hoose· 1 shared A. M. Michie· 1 shared C. Munje· 1 shared E. Gomez-Castaneda· 1 shared H. G. Jorgensen· 1 shared H. Jorgensen· 1 shared J. Bittencourt-Silvestre· 1 shared L. Hopcroft· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Hoose

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2 papers

Recent·Most cited

Tyrosine kinase inhibitor independent gene expression signature in CML offers new targets for LSPC eradication therapy

E. Gomez-Castaneda, S. Rogers, C. Munje, J. Bittencourt-Silvestre, M. Copland, D. Vetrie, L. Hopcroft, H. Jorgensen, T. Holyoake

The paper identifies TKI-independent genes in CML LSC that could be targeted for new therapies, showing that gemtuzumab-ozogamicin and cyclosporine A have stronger effects on CML cells than healthy cells.

Cancers · Oct 26, 2022Peer-reviewedCancer Biology

A KDM4A-PAF1-mediated epigenomic network is essential for acute myeloid leukemia cell self-renewal and survival

M. E. Massett, L. Monaghan, S. D. Patterson, N. Mannion, R. P. Bunschoten, A. Hoose, S. Marmiroli, R. M. Liskamp, H. G. Jorgensen, D. Vetrie, A. M. Michie, X. Huang

This study identifies KDM4A's role in AML by showing its regulation of an epigenomic network critical for cell survival and self-renewal, and highlights a gene signature for risk stratification.

bioRxiv · Oct 30, 2020Cancer Biology