Author

Daina Eiser

Henry M. Jackson Foundation

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetic & Genomic Medicine· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Assay:scRNA-seq· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:eQTL / molecular QTL mapping· 1

Frequent Co-Authors

Aidan O’BrieniD· 2 shared Irene J. CollinsiD· 2 shared Jason W. HoskinsiD· 2 shared Jun ZhongiD· 2 shared Katelyn E. ConnellyiD· 2 shared Laufey Thora AmundadottiriD· 2 shared Alison P. KleiniD· 1 shared Ashan DayanandaiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Aidan O’BrienORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Unique CD8+ T Cell Populations Expand during ART and Predict Delayed HIV-1 Rebound

Jie Wang, Gautam Kundu, Philip K. Ehrenberg, Aviva Geretz, Daina Eiser, Timothy Ezebuiro, Ashan Dayananda, Hiroshi Takata, Carlo Sacdalan, Somchai Sriplienchan, Nittaya Phanuphak, Suteeraporn Pinyakorn, Nelson L. Michael, Lydie Trautmann, Fredrick Sawe, , the RV254 study team

The paper identifies a unique CD8+ T cell population that expands during ART and predicts delayed HIV-1 rebound.

bioRxiv · Aug 18, 2026 · PreprintImmunology

Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas

Jason W. Hoskins, Trevor A. Christensen, Daina Eiser, Erin Char, Michael Mobaraki, Aidan O’Brien, Irene J. Collins, Jun Zhong, Minal B. Patel, Gauri Prasad, H. Efsun Arda, Katelyn E. Connelly, Laufey Thora Amundadottir

This study identifies rs9581943 as a causal variant in a PDAC GWAS signal, showing it reduces PDX1 expression and impairs stress buffering in exocrine pancreatic cells.

medRxiv · Apr 15, 2026 · PreprintGenetic & Genomic Medicine

Allelic effects on KLHL17 expression likely mediated by JunB/D underlie a PDAC GWAS signal at chr1p36.33

Katelyn E. Connelly, Katherine Hullin, Ehssan Abdolalizadeh, Jun Zhong, Daina Eiser, Aidan O’Brien, Irene J. Collins, Sudipto Das, Gerard Duncan, Stephen Jacob Chanock, Rachael Z. Stolzenberg‐Solomon, Alison P. Klein, Brian Matthew Wolpin, Jason W. Hoskins, Þorkell Andrésson, , Laufey Thora Amundadottir

The paper fine-maps a PDAC GWAS signal at chr1p36.33 and identifies KLHL17 as a target gene regulated by JunB/D, with potential implications for inflammation and PDAC risk.

medRxiv · Sep 16, 2024 · PreprintGenetic & Genomic Medicine