Author

E. Madrazo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

J. Redondo-Munoz· 2 shared R. Gonzalez-Novo· 2 shared A. De Lope· 1 shared A. de Lope-Planelles· 1 shared A. Gonzalez-Murillo· 1 shared D. Acitores· 1 shared F. Monroy· 1 shared H. Lopez-Menendez· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

J. Redondo-Munoz

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Persistent confined migration confers permanent nuclear and functional changes in migrating cells.

A. de Lope-Planelles, R. Gonzalez-Novo, E. Madrazo, H. Zamora-Carreras, V. Torrano, H. Lopez-Menendez, P. Roda-Navarro, F. Monroy, J. Redondo-Munoz

The paper shows that persistent confined migration causes permanent nuclear and functional changes in cells, including lamin B1 redistribution and altered DNA repair.

bioRxiv · Jan 5, 2023 · PreprintCell Biology

Targeting H3K4 methylation as a novel therapeutic strategy against tumor infiltration and nuclear changes of acute lymphoblastic leukemia cells.

R. Gonzalez-Novo, A. De Lope, A. Gonzalez-Murillo, E. Madrazo, D. Acitores, M. Garcia de Lacoba, M. Ramirez, J. Redondo-Munoz

This study shows that targeting H3K4 methylation via WDR5 inhibition reduces leukemia cell invasion in vitro and tissue infiltration in vivo, revealing a novel therapeutic strategy for acute lymphoblastic leukemia.

bioRxiv · Jun 16, 2022 · PreprintCancer Biology