Author

E. Serra

Primary ORCID profile (0000-0003-0581-7282 · 3 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

E. SerraiD3 papers · Aarhus University

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:genetic perturbation / CRISPR· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Negro· 2 shared B. Gel Moreno· 2 shared E. Castellanos· 2 shared H. Mazuelas· 2 shared I. Blanco· 2 shared I. Rosas· 2 shared M. Carrio· 2 shared A. Pezza· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

A. Negro

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Unbalancing cAMP and Ras/MAPK pathways as a therapeutic strategy for cutaneous neurofibromas

H. Mazuelas, M. Magallon-Lorenz, I. Uriarte-Arrazola, A. Negro, I. Rosas, I. Blanco, E. Castellanos, C. Lazaro, B. Gel Moreno, M. Carrio, E. Serra

This study identifies a therapeutic strategy for cutaneous neurofibromas by combining a MEKi and a cAMP elevator to disrupt SC-FB crosstalk and inhibit tumor growth.

JCI Insight · Feb 8, 2024Peer-reviewedCancer Biology

MERLIN-DEFICIENT iPSCs AS AN IN VITRO MODEL SYSTEM FOR STUDIYNG NF2 PATHOGENESIS

N. Catasus, M. Torres-Martin, A. Negro, B. Kuebler, I. Rosas, G. Casals, H. Mazuelas, F. Roca-Ribas, E. Amilibia, B. Aran, A. Veiga, A. Raya, B. Gel Moreno, I. Blanco, E. Serra, , E. CASTELLANOS

The paper generates and characterizes NF2-deficient iPSCs to study NF2 pathogenesis, showing they can model merlin-deficient Schwann cell features in vitro.

bioRxiv · Dec 14, 2022 · PreprintCell Biology

Essential bromodomain TcBDF2 as a drug target against Chagas disease

A. Pezza, L. E. Tavernelli, V. L. Alonso, V. G. Perdomo, R. C. Gabarro, R. Prinjha, E. Rodriguez Araya, I. Rioja, R. Docampo, F. Calderon, J. J. Martin, E. Serra

This paper identifies TcBDF2 as an essential bromodomain protein in Trypanosoma cruzi, showing its role in parasite survival and pathogenesis, and demonstrates its potential as a drug target.

bioRxiv · Jan 25, 2022Cell Biology