Author

E. Styring

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

D. Baumhoer· 2 shared D. C. J. Spierings· 2 shared F. Foijer· 2 shared J. Nilsson· 2 shared K. H. Nord· 2 shared K. H. Saba· 2 shared L. Magnusson· 2 shared M. Nathrath· 2 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

D. Baumhoer

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2 papers

Recent·Most cited

CDK4 is co-amplified with either TP53 promoter gene fusions or MDM2 through distinct mechanisms in osteosarcoma

K. H. Saba, V. Difilippo, E. Styring, J. Nilsson, L. Magnusson, H. van den Bos, D. C. J. Spierings, F. Foijer, M. Nathrath, F. Haglund de Flon, D. Baumhoer, K. H. Nord

This study identifies four subgroups of osteosarcomas based on genomic and transcriptomic analysis, revealing distinct mechanisms of CDK4 amplification in relation to MDM2 or TP53 alterations.

bioRxiv · Mar 13, 2024 · PreprintCancer Biology

Dysregulated gene expression through TP53 promoter swapping in osteosarcoma

K. H. Saba, V. Difilippo, M. Kovac, L. Cornmark, L. Magnusson, J. Nilsson, H. H. van den Bos, D. C. J. Spierings, M. Bidgoli, T. Jonson, V. P. Sumathi, O. Brosjo, J. Staaf, F. Foijer, E. Styring, , K. H. Nord

This study investigates how TP53 promoter swapping in osteosarcoma leads to dysregulated gene expression and oncogenic activation.

bioRxiv · Apr 20, 2020 · PreprintCancer Biology