Author

E. Tramontano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Corona· 1 shared A. Ferraro· 1 shared C. Montesano· 1 shared C. Nigri· 1 shared C. Vornholt· 1 shared D. Inverso· 1 shared D. Marotta· 1 shared E. Bono· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

A. Corona

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Nirmatrelvir treatment blunts the development of antiviral adaptive immune responses in SARS-CoV-2 infected mice

V. Fumagalli, P. Di Lucia, M. Rava, D. Marotta, E. Bono, S. Grassi, L. Donnici, R. Cannalire, I. Stefanelli, A. Ferraro, F. Esposito, E. Pariani, D. Inverso, C. Montesano, S. Delbue, , M. Iannacone

This study shows that nirmatrelvir treatment in SARS-CoV-2 infected mice reduces antibody and T cell responses, leading to weaker memory cell recruitment upon secondary challenge.

bioRxiv · Nov 22, 2022Immunology

RNA-seq transcriptome analysis reveals LTR-retrotransposons modulation in Human Peripheral Blood Mononuclear Cells (PBMCs) after in vivo Lipopolysaccharides (LPS) injection

M. P. Pisano, O. Tabone, M. Bodinier, N. Grandi, J. Textoris, F. Mallet, E. Tramontano

The paper analyzes RNA-seq data from PBMCs before and after LPS exposure to identify LTR-retrotransposon expression and modulation, finding 8.4% of loci expressed and 4,607 modulated.

bioRxiv · Dec 20, 2019Microbiology

Mutation of Ebola virus VP35 Ser129 uncouples interferon antagonist and replication functions

M. J. Morwitzer, A. Corona, L. Zinzula, E. Fanunza, C. Nigri, S. Distinto, C. Vornholt, V. Kumar, E. Tramontano, S. P. Reid

This study shows that phosphorylation of VP35 Ser129 modulates its functions, uncoupling interferon inhibition from replication, and identifies it as a potential therapeutic target.

bioRxiv · Aug 7, 2019 · PreprintMicrobiology