Author

F. Marzetta

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

C. Roger· 1 shared D. Ziegler· 1 shared F. Preitner· 1 shared I. Lopez-Mejia· 1 shared J. Blanco Fernandez· 1 shared J. Castillo-Armengol· 1 shared J. M. Vanacker· 1 shared k. Huber· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

C. Roger

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1 paper

Recent·Most cited

CDK4 deletion in mice prevents fat accumulation and increases endurance capacity through activation of estrogen related receptor (ERR)driven oxidative metabolism in skeletal muscle

V. Barquissau, N. Zanou, S. Geller, J. Castillo-Armengol, F. Marzetta, k. Huber, D. Ziegler, I. Lopez-Mejia, J. Blanco Fernandez, C. Roger, N. Guex, F. Preitner, J. M. Vanacker, L. Fajas

This study shows that CDK4 deletion in mice improves endurance and reduces fat accumulation by enhancing ERR-driven oxidative metabolism in skeletal muscle.

bioRxiv · Mar 3, 2022 · PreprintPhysiology