Author

J. C. Garbe

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

C. L. Bishop· 2 shared J. Koh· 2 shared M. P. Philpott· 2 shared M. R. Stampfer· 2 shared A. Gutierrez Del Arroyo· 1 shared B. K. Hughes· 1 shared B. Veytsman· 1 shared D. Yllanes· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

C. L. Bishop

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Author profile →

3 papers

Recent·Most cited

Configurational entropy is an intrinsic driver of tissue structural heterogeneity

V. Srivastava, J. L. Hu, J. C. Garbe, B. Veytsman, S. F. Shalabi, D. Yllanes, M. Thomson, M. A. LaBarge, G. Huber, Z. J. Gartner

The paper uses human mammary organoids to show that configurational entropy drives tissue structural heterogeneity by linking structural parameters to molecular and microenvironmental factors.

bioRxiv · Jul 1, 2023 · PreprintBioengineering

Early growth response 2 (EGR2) is a novel regulator of the senescence program

E. J. Tyler, A. Gutierrez Del Arroyo, R. Wallis, B. K. Hughes, J. C. Garbe, M. R. Stampfer, J. Koh, R. Lowe, M. P. Philpott, C. L. Bishop

This study identifies EGR2 as a novel regulator of senescence by activating the ARF and p16 promoters and reversing the senescent phenotype upon its ablation.

bioRxiv · Sep 30, 2020Cell Biology

Ribosomal stress-induced senescence as a novel pro-senescence strategy for p16 positive basal-like breast cancer

M. Moore, L. Gammon, S. Dreger, J. Koh, J. C. Garbe, M. R. Stampfer, M. P. Philpott, L. J. Jones, C. L. Bishop

This study identifies ribosomal proteins as pro-senescence targets in p16 positive basal-like breast cancer, showing their role in inducing stable cell cycle arrest and poor prognosis.

bioRxiv · Nov 14, 2018 · PreprintCancer Biology