Author

K. Wilder-Romans

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

C. A. Lyssiotis· 2 shared D. R. Wahl· 2 shared A. C. Kimmelman· 1 shared A. J. Scott· 1 shared A. L. Elaimy· 1 shared A. Lin· 1 shared A. Myers· 1 shared A. Obrien· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

C. A. Lyssiotis

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2 papers

Recent·Most cited

Cancer BiologybioRxivApr 12, 2023

GTP signaling links metabolism, DNA repair, and responses to genotoxic stress

W. Zhou, Z. Zhao, A. Lin…·D. R. Wahl

This study identifies a GTP-mediated signaling pathway linking metabolism to DNA repair and resistance to genotoxic stress in glioblastoma and non-malignant cells.

DOI 10.1158/2159-8290.cd-23-0437

humanmouse

Cancer BiologyCancer & MetabolismPeer-reviewedJan 2, 2020

Tissue of Origin Dictates GOT1 Dependence and Confers Synthetic Lethality to Radiotherapy

B. S. Nelson, L. Lin, D. M. Kremer…·C. A. Lyssiotis

The paper shows that pancreatic ductal adenocarcinoma (PDA) is more dependent on GOT1 than colorectal cancer (CRC) and that GOT1 inhibition synergizes with radiotherapy to inhibit PDA growth.

DOI 10.1186/s40170-019-0202-2

humanbulk RNA-seq