Author

M. Alpaugh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

A. C. Anderson· 1 shared A. Droit· 1 shared A. P. Yeola· 1 shared C. Joly-Beauparlant· 1 shared C. S. Moore· 1 shared H. Lassmann· 1 shared J. B. Williams· 1 shared J. Baillargeon· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. C. Anderson

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Male sex chromosomal complement exacerbates the pathogenicity of Th17 cells in a chronic model of CNS autoimmunity.

P. M. I. A. Doss, J. Baillargeon, A. P. Yeola, J. B. Williams, M. Leclercq, C. Joly-Beauparlant, M. Alpaugh, A. C. Anderson, A. Droit, H. Lassmann, C. S. Moore, M. Rangachari

This study shows male sex chromosomal complement increases Th17 cell pathogenicity in CNS autoimmunity, independent of hormones, through downregulation of Jarid1c.

bioRxiv · Jun 13, 2019 · PreprintImmunology