Author

C. Joly-Beauparlant

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Droit· 3 shared A. P. Yeola· 2 shared C. S. Moore· 2 shared J. Baillargeon· 2 shared M. Rangachari· 2 shared P. M. I. A. Doss· 2 shared A. Bairam· 1 shared A. C. Anderson· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersImmunology

A. Droit

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3 papers

Recent·Most cited

ImmunologyNature CommunicationsPeer-reviewedJun 26, 2024

Myelin-reactive B cells exacerbate the severity of CD4+ T cell-driven CNS autoimmunity in an IL-23-dependent manner.

M. R. Fazazi, P. M. I. A. Doss, R. Pereira…·M. Rangachari

This study shows that MOG-specific B cells worsen CD4+ T cell-driven CNS autoimmunity by promoting Th17 and Tph cell accumulation via IL-23.

DOI 10.1038/s41467-024-49259-0

mousebulk RNA-seq

𝕏 PostBluesky

PhysiologybioRxivMay 24, 2023

Concomitant effects of orchiectomy and intermittent hypoxia onhepatic oxidative stress, expression of flavin-containingmonooxygenases and transcriptomic profile in mice.

G. Ganouna Cohen, F. Marcouiller, C. Joly-Beauparlant…·V. Joseph

This study examines how orchiectomy and intermittent hypoxia interact to affect hepatic oxidative stress and gene expression in mice.

DOI 10.1111/apha.14089

mousebulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintJun 13, 2019

Male sex chromosomal complement exacerbates the pathogenicity of Th17 cells in a chronic model of CNS autoimmunity.

P. M. I. A. Doss, J. Baillargeon, A. P. Yeola…·M. Rangachari

This study shows male sex chromosomal complement increases Th17 cell pathogenicity in CNS autoimmunity, independent of hormones, through downregulation of Jarid1c.

DOI 10.1101/640904

mousebulk RNA-seq

𝕏 PostBluesky