Author

M. Rangachari

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

J. Baillargeon· 3 shared P. M. I. A. Doss· 3 shared A. Droit· 2 shared A. P. Yeola· 2 shared C. Joly-Beauparlant· 2 shared C. S. Moore· 2 shared I. Akbar· 2 shared A. C. Anderson· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersImmunology

J. Baillargeon

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Myelin-reactive B cells exacerbate the severity of CD4+ T cell-driven CNS autoimmunity in an IL-23-dependent manner.

M. R. Fazazi, P. M. I. A. Doss, R. Pereira, N. Fudge, A. Regmi, C. Joly-Beauparlant, I. Akbar, A. P. Yeola, B. Mailhot, J. Baillargeon, P. Grenier, N. Bertrand, S. Lacroix, A. Droit, C. S. Moore, , M. Rangachari

This study shows that MOG-specific B cells worsen CD4+ T cell-driven CNS autoimmunity by promoting Th17 and Tph cell accumulation via IL-23.

bioRxiv · Sep 6, 2023Immunology

Serpine1 negatively regulates Th1 cell responses in experimental autoimmune encephalomyelitis.

I. Akbar, R. Tang, J. Baillargeon, A.-P. Roy, P. M. I. A. Doss, C. Zhu, V. K. Kuchroo, M. Rangachari

This study shows Serpine1 inhibits Th1 cell pathogenicity in EAE by promoting Tim-3 expression and function.

bioRxiv · Jul 13, 2023 · PreprintImmunology

Carcinogen induced expansion of atypical B cells and pre-treatment tumor adjacent tertiary lymphoid structures associate with poor response to BCG in non-muscle invasive bladder cancer

P. Yolmo, S. Rahimi, S. Chenard, G. Conseil, D. Jenkins, I. Emon, J. Hamilton, M. Xu, M. Rangachari, E. Michaud, J. Mansure, D. M. Berman, D. R. Siemens, M. Koti

The paper shows that atypical B cells and pre-treatment tumor-associated tertiary lymphoid structures are linked to poor BCG response in NMIBC, with BBN-induced inflammation promoting ABCs in female mice.

bioRxiv · Dec 30, 2022 · PreprintCancer Biology

Male sex chromosomal complement exacerbates the pathogenicity of Th17 cells in a chronic model of CNS autoimmunity.

P. M. I. A. Doss, J. Baillargeon, A. P. Yeola, J. B. Williams, M. Leclercq, C. Joly-Beauparlant, M. Alpaugh, A. C. Anderson, A. Droit, H. Lassmann, C. S. Moore, M. Rangachari

This study shows male sex chromosomal complement increases Th17 cell pathogenicity in CNS autoimmunity, independent of hormones, through downregulation of Jarid1c.

bioRxiv · Jun 13, 2019 · PreprintImmunology